Eyelid Swelling

Eyelid Swelling Ppts and published articles

Neonatal Conjunctivitis
Patricia Wang,
http://www.medicine.nevada.edu/

Orbital Tumors
Michael Underbrink, MD, Shawn Newlands, MD, PhD
http://www.utmb.edu

Ophthalmologic Emergencies
http://emed.stanford.edu

Injuries to the Face, Eye, and Throat
http://faculty.sgc.edu

Diseases of the Eye
Casey Conway , Jeannie Stall
https://bbmedia.dmacc.edu

Eyelid Swelling Caused By Lymphoma
John S. Kennerdell, MD
http://content.lib.utah.edu

Facial Injuries
http://wweb.uta.edu

Hypothyroidism - Signs and Symptoms
Classic Teaching
http://www.meddean.luc.edu

Prevention of Injuries to the Head, Face, Eyes, Ears, Nose, and Throat
http://www2.fiu.edu

Natural Facial Markings Associated With the Mouth
http://www.mccc.edu

Physiognomical Form of the Eye
http://www.mccc.edu

Pathogenic Human Helminthes and Arthropods
http://iws.collin.edu

Grand Rounds
Jeffrey D. Colburn, M.D.
http://www.mc.vanderbilt.edu

The Special Senses
http://faculty.ccri.edu 

100 Published articles on Eyelid Swelling

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Optic Nerve Hypoplasia

Direct Ophthalmoscope
http://medicine.missouri.edu/

Causes & Functional Implications of Visual Impairment
http://streaming.osu.edu

Vision Loss
http://www.cte.ku.edu

Cortical Visual Impairment and Blindness Functional Implications and Rehabilitation
Kia B. Eldred, OD, FAAO
http://www.hadley.edu

Facts for Native Hawaiians about Smoking and Pregnancy
http://kapalama.ksbe.edu

Brainstem glioma
http://neuroradiology.rad.jhmi.edu

Failed Eye Exam
Rochelle Cochrane, M.D., Tracy Crnic, M.D.
https://www.ttuhsc.edu

Glaucoma
http://www.opt.uab.edu

Optic Nerve Hypoplasia
Julie Greenlee, Angela Howe
http://www.faculty.sfasu.edu 
 
180 Published articles on Optic Nerve Hypoplasia

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Eyelid Swelling

Facial Injuries Eye/Nose/Face
Eye - purpose/anatomy
http://teacher.edmonds.wednet.edu

Eyes
http://w3.palmer.edu

Natural Facial Markings Associated With the Mouth
http://www.mccc.edu

Parasitic Worms
http://iws.collin.edu

Evaluation of Eye Pathologies
Orthopedic Assessment III – Head, Spine, and Trunk with Lab
http://www2.fiu.edu

Soft-Tissue Injuries
http://ww2.shoreline.edu

Dermatomyositis
Emily O. Jenkins MD
https://medicine.med.unc.edu

The eyes have it. Ophthalmologic emergencies
Cecilia Guthrie, MD
http://www.pediatrics.emory.edu

Eye, Ear & Maxillofacial Pathologies
Kimberly Lakhan, PA-C
http://www.d.umn.edu

Case Scenario - What’s Your Diagnosis??
http://www.bcm.edu

The Special Senses
http://faculty.ccri.edu

Ophthalmologic Emergencies
http://emed.stanford.edu

Facial Injuries
http://wweb.uta.edu

Injuries to the Eye
http://faculty.sgc.edu

Orbital Tumors
Michael Underbrink, MD, Shawn Newlands, MD, PhD
http://www.utmb.edu

The Head, Face, Eyes, Ears, Nose, and Throat
http://www2.fiu.edu

Parasitic Infestations, Stings & Bites
http://www.atsu.edu

Prevention of Injuries to the Head, Face, Eyes, Ears, Nose and Throat
http://www.lscc.edu


50 Published articles on Eyelid Swelling

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Periorbital oedema

Diffuse Eyelid Disease
http://web.squ.edu.om/med-Lib/

Sinusitis
Rhonda Lesniak
http://www.fasn.net/documents/articles/SINUSITIS.ppt

Pediatric Rhinosinusitis
Sarah Rodriguez, MD, Matthew Ryan, MD
http://www.utmb.edu/otoref/grnds/

Infectious Mononucleosis
Adam Stern, PGY III
http://www.med.unc.edu/

Thyroid Eye Disease
aka Thyroid Associated Ophthalmopathy
http://www.icoph.org/med/

Renal SAQ - answers
David Robinson
http://www.yorksandhumberdeanery.nhs.uk/emergencymedicine

Head Injury
Saurabh Sinha
http://www.dcn.ed.ac.uk/

Nasal fractures Trauma to nose
http://sun025.sun.ac.za

Understanding Mold Related Illnesses
http://www.drcordas.com/

My Aching Joints!
Jon Roebuck CPT MC
http://www.usuhs.mil/med/

Hypothyroidism - Signs and Symptoms Classic Teaching
http://www.meddean.luc.edu/

Thyroid Disease -Intern Conference
http://www.med.unc.edu/medicine

Thyroid disorders in everyday care
Chris Vreeland
http://enp-network.s3.amazonaws.com

Trichinellosis
Causal Agents
http://api.ning.com/


13 Published articles on Periorbital oedema

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Pterygium

Update on Pterygium Therapy
Jay C. Bradley, MD, David L. McCartney, MD
https://www.ttuhsc.edu

Diseases of the Eye
http://www.uams.edu

Red Eye and Ocular Trauma
Harold E Cross MD PhD
http://www.eyes.arizona.edu

The Irritated Eye
Liana Al-Labadi, O.D.
http://elearning.najah.edu

Eye Blocks and Airway Blocks
Vincent Conte, MD
http://chua2.fiu.edu

Transition from School to Adulthood: New Horizons for Youth with Disabilities
Auburn, Alabama, Paul Wehman, Ph.D.
https://fp.auburn.edu

Some Genodermatoses and Acquired Syndromes
Rick Lin, DO MPH
http://www.atsu.edu

Genodermatosis
Boris Ioffe, D.O.
http://www.atsu.edu

Nuclear Radiation
http://chemistry.csudh.edu

Beach Safety
http://www.canyons.edu

Ocular Emergencies
Lianne Beck, MD

http://www.fpm.emory.edu

New Surgical Technique: Use of Indocyanine Green and Forced Hydrodissection Fortotal Anterior Lamellar Keratoplasty
Thomas John, MD
http://www.stritch.luc.edu

X-Linked Juvenile Retinoschisis
Laura S. Gilmore, MD, MS
http://www.ttuhsc.edu


276 free full text published articles on Pterygium

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Central Retinal Artery Occlusion

Ocular Urgencies and Emergencies
http://www.opt.uab.edu

Eye Spy - A Case of Acute Vision Loss
Tony S. Chang, MD
http://www2.medicine.wisc.edu

Selected cases in ophthalmic pathology
Amy Lin, M.D.
https://www.med.illinois.edu

Case Studies in Ophthalmology
http://ophthalmology.wustl.edu

Transient Monocular Blindness
Shirley H. Wray, M.D., Ph.D.,
http://novel.utah.edu

Direct Ophthalmoscope
http://medicine.missouri.edu

Systemic Diseases and the Eye
http://www.uthsc.edu

Hemi Central Retinal Vein Occlusion
John Nicolau, M.D., Leo Dominguez, M.D.
http://www.ttuhsc.edu

X-Linked Juvenile Retinoschisis
Laura S. Gilmore, MD, MS
http://www.ttuhsc.edu

The Visual System
Dr. Sue Semple-Rowland
http://users.phhp.ufl.edu

Lesions affecting pupil responses
http://users.phhp.ufl.edu

Eye Emergencies
Nikki Waller
https://www.med.unc.edu

Visual System Lab
Jen Martindale
http://www-scf.usc.edu

Ophthalmologic Emergencies
http://emed.stanford.edu
384 Published articles on Central Retinal Artery Occlusion

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Preseptal and Orbital Cellulitis

Anatomy of Head and Neck Infections
Angelia Natili, MD, Susan McCammon, MD
http://www.utmb.edu

Complications of Rhinosinusitis
Viet Pham, M.D., Patricia Maeso, M.D.
http://www.utmb.edu

Rhinology Review
Marilene B. Wang, MD, FACS
http://headandnecksurgery.ucla.edu

Eye Emergencies
Nikki Waller
https://www.med.unc.edu

ENT Infections
Dr. Patamasucon
http://www.medicine.nevada.edu

Periorbital Cellulitis
Karen R Brungard MD
http://www2.medicine.wisc.edu

Clinical Problem Solving
Mark Stafford M.D., Stanford Massie M.D.
http://www.uab.edu

Red Eye and Ocular Trauma
Harold E Cross MD PhD
http://www.eyes.arizona.edu

The Red Eye
Charlise A. Gunderson, M.D.
http://www.utmb.edu

Primary Care for the Red Eye
Alice L. Bashinsky, M.D., Phillip C. Hoopes, Jr, M.D.
http://intmedweb.wfubmc.edu

Otitis
John H. Isaacs, M.D.
http://www.mgm.ufl.edu

Systemic Diseases and the Eye
http://www.uthsc.edu

Antibiotic Choices for Infections which Require Hospitalization
Rodolfo E. Bégué, MD
http://www.medschool.lsuhsc.edu

Case Presentation
http://www.ent.uci.edu

Cellulitis
http://www2.medicine.wisc.edu

Case Conference
Yvonne L. Carter, MD
http://www.med.unc.edu


Published articles

Preseptal orbital cellulitis following oral trauma.

Ganesh A, Venugopalan P.

J Pediatr Ophthalmol Strabismus. 2000 Sep-Oct;37(5):315-7.

Ectopic cilia and preseptal orbital cellulitis.

Riffle JE.

Am J Ophthalmol. 1984 Jul 15;98(1):119-20.

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Fluorescein angiography

Fluorescein angiography - A Comparison between Fluorescein Angiography and Optical Coherence Tomography
KoriAnne Elkins Galler, M.D.
Fluorescein angiography.ppt

Ophthalmic Applications of Fluorescence
Ophthalmic Applications of Fluorescence.ppt

Medicare Coding and Billing
Medicare Coding and Billing.ppt

Screening For Retinopathy & Nephropathy
Prof.V.Mohan.,M.D.,Ph.D.,D.Sc
Screening For Retinopathy & Nephropathy.ppt

Hemi Central Retinal Vein Occlusion
John Nicolau, M.D., Leo Dominguez, M.D.
Hemi Central Retinal Vein Occlusion.ppt

Macular Degeneration
Macular Degeneration.ppt

Age-Related Macular Degeneration
By Tom Boyles
Age related macular_degeneration_09.ppt

Diseases of Eye
Diseases of Eye.ppt

Primary Intraocular Lymphoma – Uveitis Masquerade Syndrome
Saiama N. Waqar, MD
Uveitis Masquerade Syndrome .ppt

Disease-Oriented Evaluation of Dual-Bootstrap Retinal Image Registration
Chia-Ling Tsai, Charles V. Stewart, Badrinath Roysam
charlene.poster.ppt

Fluorescein angiography
Fluorescein angiography.ppt

The Sensory System
The Sensory System_lecture.ppt

Fluorescein Angiogram
Alethea Hein
Fluorescein Angiogram.ppt

Epiretinal Membranes, CME and Macular Holes
Laura S. Gilmore, MD
Epiretinal Membranes, CME and Macular Holes.ppt

Phytochemicals and Ocular Disease
Paul S. Bernstein, MD, PhD
Phytochemicals and Ocular Disease.ppt
87 Free full text articles

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Macular Degeneration

What is Macular Degeneration?
Macular Degeneration is a group of progressive eye conditions which involve deterioration of the macula, the central region of the retina.

Age-Related Macular Degeneration
By Tom Boyles, Dustin Broadus, Lori Buckle, Constance Marsh, and April Seybold
http://www.faculty.sfasu.edu/munromicha/spe516/macular_degeneration_09.ppt

Age-Related Macular Degeneration
Sabrina Carter & Kori Eakin
http://www.faculty.sfasu.edu/munromicha/spe516/amd9.ppt

Antioxidants and Nutrients in the Treatment and Prevention of Age-Related Macular Degeneration
Michael B. Gorin, M.D. Ph.D.
http://www.healthyaging.pitt.edu/seminars2006/nutritionsymposium/gorin-Nutrition-AMDlecture.ppt

Age Related Macular Degeneration
http://www.fpm.emory.edu/Family/didactics/powerpint/AFP%20Journal%20Review%205-15-08(2).ppt

Macular Degeneration
http://www.mccc.edu/~martinl/documents/M21fVISUALALTERATIONSmaculardegeneration.ppt

Advances in the Treatment of Age-Related Macular Degeneration (AMD)
Michael E. Helm, PA-S
AMD.ppt

Visual Impairment
Visual Impairment.ppt

Career Counseling for Clients who Are Blind or Visually Impaired
Lauri Dishman, M.A., LCPC
Career_Counseling_for_People_with_Vision_Loss.ppt

 Eye disorders
Eye disorders.ppt

Telescopic Implants
Joseph Cullen
Telescopic Implants.ppt

The Impact of Cataract Surgery in Patients with Low Vision
Clinical Research Methods
Irene C. Kuo, MD
The Impact of Cataract Surgery in Patients with Low Vision.ppt

Making Sense of Sensory Losses as We Age
Dena Kemmet, M.S. and Sean Brotherson, Ph.D.
Making Sense of Sensory Losses as We Age.ppt

The Eyes Have It!
Eyes_have_it/can_you_tell_the_difference.ppt

Vision II
S. Buckley RN, MSN
visiontwo.ppt
200 scholarly publications free access

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Vernal keratoconjunctivitis VKC 50 scholarly articles free access

Vernal keratoconjunctivitis (VKC) or Spring catarrh is a recurrent, bilateral, and self limiting inflammation of conjunctiva, having a periodic seasonal incidence.

1.    Images in clinical medicine. Vernal keratoconjunctivitis.

Homme J, Wiswell J.

N Engl J Med. 2012 Feb 16;366(7):647. No abstract available.

2.    Direct fluorescent antibody assay and polymerase chain reaction for the detection of Chlamydia trachomatis in patients with vernal keratoconjunctivitis.

Nishiwaki-Dantas MC, de Abreu MT, de Melo CM, Romero IL, Neto RB, Dantas PE.

Clinics (Sao Paulo). 2011;66(12):2013-8.

3.    A clinical study to assess the efficacy of Triyushnadi Anjana in Kaphaja Abhishyanda with special reference to vernal keratoconjunctivitis.

Dhiman KS, Sharma G, Singh S.

Ayu. 2010 Oct;31(4):466-72.

4.    Childhood chronic anterior uveitis associated with vernal keratoconjunctivitis (VKC): successful treatment with topical tacrolimus. Case series.

Taddio A, Cimaz R, Caputo R, de Libero C, Di Grande L, Simonini G, Mori F, Novembre E, Pucci N.

Pediatr Rheumatol Online J. 2011 Nov 2;9(1):34.

5.    Association of hemopexin in tear film and conjunctival macrophages with vernal keratoconjunctivitis.

Pong JC, Chu CY, Li WY, Tang LY, Li L, Lui WT, Poon TC, Rao SK, Lam DS, Wang CC, Pang CP.

Arch Ophthalmol. 2011 Apr;129(4):453-61.

6.    Are cutaneous hypersensitivity tests to inhalant allergens a severity marker for vernal keratoconjunctivitis?

Augusto de Oliveira L, Mallozi MC, Sole D, Freitas D, Sousa LB, Mannis MJ.

Arq Bras Oftalmol. 2007 Nov-Dec;70(6):991-5.

7.    Conjunctival Inclusion Cysts in Long-standing Chronic Vernal Keratoconjunctivitis.

Lee SW, Lee SC, Jin KH.

Korean J Ophthalmol. 2007 Dec;21(4):251-4.

8.    Natural killer cells in vernal keratoconjunctivitis.

Lambiase A, Normando EM, Vitiello L, Micera A, Sacchetti M, Perrella E, Racioppi L, Bonini S, Bonini S.

Mol Vis. 2007 Aug 30;13:1562-7.

9.    [Efficacy of topical 2% cyclosporine A as a steroid-sparing agent in steroid-dependent vernal keratoconjunctivitis].

Doan S, Gabison E, Abitbol O, Gatinel D, Chast F, Hoang-Xuan T.

J Fr Ophtalmol. 2007 Sep;30(7):697-701. French.

10.  Nerve growth factor has a modulatory role on human primary fibroblast cultures derived from vernal keratoconjunctivitis-affected conjunctiva.

Micera A, Lambiase A, Stampachiacchiere B, Sgrulletta R, Normando EM, Bonini S, Bonini S.

Mol Vis. 2007 Jun 21;13:981-7.

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Epiretinal membrane - macular pucker Ppts and scholarly articles

An epiretinal membrane (also known as macular pucker) is a thin sheet of fibrous tissue that can develop on the surface of the macular area of the retina and cause a disturbance in vision.

Epiretinal membranes in Macular Dysfunction
Epiretinal membranes in Macular Dysfunction GrandRounds3.ppt

Epiretinal membranes in Macular Dysfunction
X-Linked Juvenile Retinoschisis
Laura S. Gilmore, MD, MS
X-Linked Juvenile Retinoschisis GrandRounds.ppt

Epiretinal membranes in Macular Dysfunction
Unexplained Visual Loss
Laura S. Gilmore, MD
Unexplained Visual Loss GrandRounds.ppt

Epiretinal Membranes, CME and Macular Holes
Laura S. Gilmore, MD
CME and Macular Holes GrandRounds3.ppt

Diabetic Retinopathy
Steven Sanislo, M.D.
Diabetic Retinopathy.ppt

Retinal Bioengineering
Retinal Bioengineering.ppt

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Internuclear Ophthalmoplegia

 
Bilateral  Internuclear Ophthalmoplegia
http://novel.utah.edu/Wray/PPT/Bilateral_Internuclear_Ophthalmoplegia.ppt

Internuclear  Ophthalmoplegia in Childhood
Shirley  H. Wray, M.D., Ph.D
http://novel.utah.edu/Wray/PPT/Internuclear_Ophthalmoplegia_in_Childhood_guest_lecture.ppt

Internuclear Ophthalmoplegia in MS
David  Jho
http://www2.uic.edu/stud_orgs/hon/aoa/M1reviews/Neuroanatomy.ppt

Internuclear Ophthalmoplegia
Mily Kannarkat
http://www.med.unc.edu/medicine/web/4.22.09%20Kannarkat.%20Internuclear%20ophthalmoplegia.ppt

Internuclear Ophthalmoplegia: MLF lesion
http://www.clas.ufl.edu/users/sgriff/ENG&VNG-Occulomotor%20Tests.ppt

Top Ten Things You Should Know About The Oculomotor System
http://www.opt.uab.edu/class2011/1st%20year/NeuroAnatomy/oculomotor.ppt

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Anisocoria Ppt Lecture Notes

Anisocoria, or unequal pupil sizes, is a common condition. The varied causes have implications ranging from life threatening to completely benign.

Pupil  Evaluation
Walter  Huang, OD
Pupil Evaluation.ppt

Taking the Mystery out of Abnormal Pupils
Brad Sutton, O.D., F.A.A.O.
Taking-the-mystery-pupils.ppt

Neuro-ophthalmology
Abdulrahman  Al-Muammar
Lectures/Neuro-ophthalmology.ppt

Pupil
Pupil.ppt

Case  Presentation - Doctor my pupils are dilated
Richard Plantive
Anisocoria.ppt

Herniation: Compartment Syndrome of the Head
Connie  Chen, MD
Herniation.ppt

Eyedrop testing in Horner Syndrome
Eugene F. May, MD
MayHorners.ppt.ppt

Evaluation  of Eye Pathologies
Eye Evaluation.ppt

Eye Injury Assessment
Eval/eye.ppt

Scopolamine
Julia Bedell, Laci Click, Meredith Barr
Scopolamine.ppt

The Pupil
Pupil.ppt
View PDFs here

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Toxoplasmosis Ppts and recent articles

Toxoplasmosis is an infectious disease caused by protozoan parasite Toxoplasma gondii.

Major  Human Parasites
Major human Protozoa.ppt

Eye manifestations  during pregnancy
By Dr  Mohamed Abusharifa, MD
Eye-manifestations-during-pregnancy.ppt

Ocular  Toxoplasmosis: A Common Manifestation of a Not-So-Common Disease
Mehdi  Shahidi, MD, Vidya  Sundareshan, MD, MPH
Ocular Toxoplasmosis .ppt

Toxoplasmosis 
by Pam  Holmes, Christy Stalcup, and Pam Yarborough
Toxoplasmosis.ppt

New Molecular  Based Methods of Diagnosis
Molecular Methods of Diagnosis.ppt

Toxoplasmosis
http://www.uic.edu/depts/ci/neonat05/Handouts/TH6-CMV05-Pyati.ppt

Infectious  Mononucleosis
http://cls.umc.edu/COURSES/CLS311/InfMono.ppt

Systemic  Diseases and the Eye
Systemic Diseases and the Eye

Anti-protozoal  drugs
http://elearning.najah.edu/OldData/pdfs/4518antiparasitics%201.ppt

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Extraocular muscles Ppts and Latest 50 Published articles

Extraocular muscles: The extraocular muscles are the six muscles that control the movements of the (human) eye (there are four in bovines). The actions of the extraocular muscles depend on the position of the eye at the time of muscle contraction.

Extraocular Muscles (EOM) & Eye movement
http://www.opt.uab.edu/class2011/1st%20year/Ocular%20Anatomy/Opt112.No23_24EOM.ppt

Evaluation and Treatment of Oculomotor Impairment
http://www.uab.edu/shrpot/Mary%20Warren/OT%20689%20Summer03/689%20Oculomotor%20lec%20A.PPT

Monitoring Operations  in the Skull Base
http://www.utdallas.edu/~amoller/iom/6_mon_skull.ppt

Identify  the elements of the bony orbit on  a skull or x-ray
http://www.emory.edu/ANATOMY/Slides/Eye%20&%20Orbit.ppt

Eye  Movement Physiology
by Frank M. Marchak, Ph.D.
http://www.wpic.pitt.edu/research/biometrics/SPR%202011%20Workshop/Eye%20Movement%20Physiology.ppt

The Muscular System: Axial Musculature
http://faculty.sdmiramar.edu/dtrubovitz/anatomy/powerpoint/ch10.ppt

Myasthenia  Gravis
by Shirley  H. Wray, M.D., Ph.D.
http://novel.utah.edu/Wray/PPT/Myasthenia_Gravis_guest_lecture.ppt

Eye Examination
http://www.uky.edu/~achan2/med/imp1/EYES_.PPT

Eye Anatomy
http://elearning.najah.edu/OldData/pdfs/3800Lecture%201-%20Eye%20Anatomy.ppt

Model  Science – The Human Eye
http://mesa.ucop.edu/staff/masme_2010/WORKSHOPS/Model_Science_The_Human_Eye/Model_Science_The_Human_Eye.ppt

Latest 50 Publications:

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Orthokeratology, Improved glare , Visual and Auditory Problems, Aniseikonia, Contact Lens Optics, Photoreceptors ppts

Orthokeratology, Improved glare , Visual and Auditory Problems, Aniseikonia, Contact  Lens Optics, Photoreceptors ppts

Laser  Eye Surgery Technology
http://web.pdx.edu/~larosaa/Applied_Optics_464-564/Projects_Presented/Hallen_Rico_LaserEyeSurgeryTechnology.ppt

Improved glare (halos and scattered light) measurement for post-LASIK surgery
http://cornea.berkeley.edu/presentations/Mopane2003/Mopane2003.ppt

Visual and Auditory Problems
http://nah.southtexascollege.edu/ADN/assets/docs/candelaria/Chapter_022%20Visual%20&%20Auditory%20Problems.ppt

Aniseikonia
http://www.opt.indiana.edu/v666/lec06_files/lec06.ppt

Contact  Lens Optics
by Melanie  A Pickett, O.D.
http://eyeatlas.medicine.iu.edu/wp/wp-uploads/2011/10/Contact_Lens_Optics_lecture.ppt

Photoreceptors
by Scott Wallace
http://www.physics.ucla.edu/~arisaka/Phys89W09/T11/Vision_T11.ppt

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Corneal ulcer, Eye Emergencies, Red Eye and Ocular Trauma ppt and publications

Corneal Melt
by Jeremy B. Wingard, MD
http://neuro.pathology.pitt.edu/webstuff/EyePathology/09Wingard%20Pathology%20Case.ppt

Red  Eye and Ocular Trauma
Harold  E Cross MD PhD
http://www.eyes.arizona.edu/Teaching/medstudents/red%20eye%20&%20trauma.ppt

Conditions Causing Low Vision
http://www.uab.edu/shrpot/Mary%20Warren/OT%20677%20Conditions%20lecture%203.PPT

Clostridium
by Katherine Leon & Dr. Edwin Ginés-Candelaria
http://wcis.mdc.edu/browse/egines/BSC2943L_THE%20BIOSCIENCE%20INTERNSHIP/THE%20BACTERIUM%20CLOSTRIDIUM%20TAENIOSPORUM/POWERPOINT/CLOSTRIDIUM.ppt

Eye  Emergencies
by Nikki  Waller
http://www.med.unc.edu/emergmed/files/Eye_Emergencies.ppt

Ocular  Urgencies and Emergencies
http://www.opt.uab.edu/opt222/Powerpoints/CLAP%202--Rev%20Ocular%20Urgencies%20and%20Emergencies.ppt

The  Red Eye
By Charlise A. Gunderson, M.D
http://www.utmb.edu/pedi/ppts/redeye.ppt

Primary  Care for the Red Eye
by Alice  L. Bashinsky, M.D., Phillip  C. Hoopes, Jr, M.D
http://intmedweb.wfubmc.edu/download/redeye.ppt

Recent publications:

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Uveitis Powerpoint Presentations and Publications

Vogt-Koyanagi-Harada Syndrome
by Laura S. Gilmore, MD, Department of Ophthalmology
http://www.ttuhsc.edu/som/ophthalmology/lectures/presentations/11.12.04GrandRoundsVKH.ppt

Primary Intraocular Lymphoma – Uveitis Masquerade Syndrome
http://hematology.wustl.edu/conferences/presentations/Waqar20090116.ppt

Unexplained Visual Loss
by Laura S. Gilmore, MD, Grand Rounds
http://www.ttuhsc.edu/som/ophthalmology/lectures/presentations/9.9.05GrandRounds.ppt

Latest 20 publication on Uveitis

1: Babu K, Kini R, Mehta R, Philips M, Subbakrishna DK, Murthy KR.
Predictors For Tubercular Uveitis: A Comparison Between Biopsy-Proven Cases of Tubercular and Sarcoid Uveitis. Retina. 2011 Dec 5. [Epub ahead of print] PubMed PMID: 22146129.


2: Peralta-Calvo J.
Vitrectomy as anti-inflammatory treatment in intermediate uveitis.
Arch Soc Esp Oftalmol. 2011 Dec;86(12):421. Epub 2011 Sep 9. English,
Spanish. PubMed PMID: 22117744.


3: Fonollosa A, Adán A.
Uveitis: a multidisciplinary approach.
Arch Soc Esp Oftalmol. 2011 Dec;86(12):393-394. English, Spanish. PubMed PMID: 22117737.


4: Oltra EZ, Morris C, Birnbaum AD, Tessler HH, Goldstein DA.
Chronic anterior uveitis in common variable immunodeficiency.
Ocul Immunol Inflamm. 2011 Dec;19(6):448-9. PubMed PMID: 22106916.


5: Erckens RJ, Mostard RL, Wijnen PA, Schouten JS, Drent M.
Adalimumab successful in sarcoidosis patients with refractory chronic non-infectious uveitis. Graefes
Arch Clin Exp Ophthalmol. 2011 Nov 27. [Epub ahead of print] PubMed PMID: 22119879.


6: Rosenzweig HL, Woods A, Clowers JS, Planck SR, Rosenbaum JT.
The NLRP3 inflammasome is active but not essential in endotoxin-induced uveitis.
Inflamm Res. 2011 Nov 26. [Epub ahead of print] PubMed PMID: 22119862.


7: Cohen AE, Assang C, Patane MA, From S, Korenfeld M; Avion Study Investigators.
Evaluation of Dexamethasone Phosphate Delivered by Ocular Iontophoresis for Treating Noninfectious Anterior Uveitis.
Ophthalmology. 2011 Nov 22. [Epub ahead of print] PubMed PMID: 22115712.


8: Willermain F, Rosenbaum JT, Bodaghi B, Rosenzweig HL, Childers S, Behrend T, Wildner G, Dick AD. Interplay between innate and adaptive immunity in the development of non-infectious uveitis. Prog Retin Eye Res. 2011 Nov 22. [Epub ahead of print] PubMed PMID: 22120610.


9: Işık M, Kılıç L, Doğan I.
Recurrent uveitis due to sildenafil usage in a patient with Behçet's disease. Rheumatol Int. 2011 Nov 18. [Epub ahead of print] PubMed PMID: 22095394.


10: Kim TW, Kang JS, Kong JM, Bae S, Yu Y, Chung H, Yu HG.
 Maturation profiles of peripheral blood dendritic cells in patients with endogenous uveitis.
Immunol Lett. 2011 Nov 15. [Epub ahead of print] PubMed PMID: 22108236.


11: Heiligenhaus A, Michels H, Schumacher C, Kopp I, Neudorf U,…
Evidence-based, interdisciplinary guidelines for anti-inflammatory treatment of uveitis associated with juvenile idiopathic arthritis.
Rheumatol Int. 2011 Nov 15. [Epub ahead of print] PubMed PMID:
22083610.


12: Gül A, Tugal-Tutkun I, Dinarello CA, Reznikov L, Esen BA, Mirza A, Scannon P, Solinger A.
Interleukin-1β-regulating antibody XOMA 052 (gevokizumab) in the treatment of acute exacerbations of resistant uveitis of Behcet's disease: an open-label pilot study.
Ann Rheum Dis. 2011 Nov 14. [Epub ahead of print] PubMed  PMID: 22084392.


13: Cantarini L, Simonini G, Frediani B, Pagnini I, Galeazzi M, Cimaz R. Treatment strategies for childhood noninfectious chronic uveitis: an update.
Expert Opin Investig Drugs. 2011 Nov 11. [Epub ahead of print] PubMed PMID: 22074394.


14: Esterberg E, Acharya NR. Corticosteroid-sparing therapy: practice patterns among uveitis specialists. J Ophthalmic Inflamm Infect. 2011 Nov 6. [Epub ahead of print] PubMed PMID: 22057810.


15: Gallagher A.
Leptospirosis in a dog with uveitis and presumed cholecystitis.
J Am Anim Hosp Assoc. 2011 Nov;47(6):e162-7. PubMed PMID: 22058365.


16: Batmanabane V, Chidambaranathan GP, Rathinam S.
Spectrum of Leptospira species identified in patients with leptospiral uveitis in an ophthalmological institute in South India.
Indian J Med Microbiol. 2011 Oct;29(4):444-5. PubMed PMID: 22120817.


17: Santos FF, Commodaro AG, Souza AV, Pinho JR, Sitnik R, Garcia C, Ribeiro AL, …
Real-time PCR in infectious uveitis as an alternative diagnosis.
Arq Bras Oftalmol. 2011 Aug;74(4):258-61. PubMed PMID: 22068852.


18: Hunter RS, Lobo AM.
Dexamethasone intravitreal implant for the treatment of noninfectious uveitis.
Clin Ophthalmol. 2011;5:1613-21. Epub 2011 Nov 11. PubMed PMID: 22140307; PubMed Central PMCID: PMC3225458.


19: Pratap DS, Lim LL, Wang JJ, Mackey DA, Kearns LS, Stawell RJ; …
The role of toll-like receptor variants in acute anterior uveitis.
Mol Vis. 2011;17:2970-7. Epub 2011 Nov 16. PubMed PMID: 22128242; PubMed Central PMCID:
PMC3224833.


20: Yang MM, Lai TY, Tam PO, Chiang SW, Chan CK, Luk FO, Ng TK, Pang CP.
CFH 184G as a genetic risk marker for anterior uveitis in Chinese females.
Mol Vis. 2011;17:2655-64. Epub 2011 Oct 13. PubMed PMID: 22065918; PubMed Central PMCID:
PMC3209418.

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OKAP Glaucoma Review

OKAP Glaucoma Review
By:Yara Catoira-Boyle MD

Introduction to Glaucoma

* What is the definition of glaucoma?
* Group of diseases that have in common a characteristic optic neuropathy associated with visual field loss for which elevated IOP is the primary risk factor
* What are the 3 factors that determine IOP?
* Rate of aqueous production by the CB, resistance to aqueous flow (Juxtacanalicular TM) and the level of EVP
* Of those factors, what is the most common cause of increased IOP?
* Increased resistance to outflow
* What is the prevalence of glaucoma in the general population?
* 1.5-2%
* What the percentage of 1st degree relatives of POAG patients will develop the disease?
* 10-15%
* How many people over 45 y/o are estimated to have glaucoma in the US? What % is bilaterally blind?
* 2.25 million / 4%
* 10 million /2%
* 1 million / 5%
* 5 million /4%
* What is the most common cause of nonreversible blindness in AA in the US?
* Glaucoma (prevalence 3-4X higher than whites)
* What are the leading causes of blindness worldwide?
* Trachoma Cataract glaucoma
* Which one is NOT a risk factor for glaucoma?
* Increased IOP and AA race
* Positive family history
* Advanced age
* Thin central cornea
* Male gender
* DM, myopia, HTN, ischemic vascular disease, arteriosclerosis are all inconclusive
* Which one is correct about the prevalence of Chronic angle closure glaucoma?
* Inuit from Arctic Asians Whites
* Men women
* Myopes hyperopes
* Younger older
* Which one is correct about heredity of glaucoma?
* Prevalence among siblings of pts is 20%
* Lifetime absolute risk at age 89 is 10 x higher for relatives of glaucoma patients
* A single gene is likely to be discovered as the culprit
* The gene responsible for mutations of the TIGR (myocilin) protein is located on chromos 5

A little genetics of glaucoma
* TIGR protein produced by TM cells was id’d in Juvenile glaucoma, and later found to affect up to 3% of OAG
* GLC1A, the gene responsible for TIGR mutations is on chromosome 1
* It seems to be an autossomal dominant inheritance of the polygenic type with late or variable age of onset, incomplete penetrance and substantial environmental influence

IOP and Aqueous Dynamics
* What is correct about the aqueous composition?
* Has less hydrogen and chloride than plasma
* Has deficit of ascorbate
* Has excess bicarbonate
* Contains lysozyme, cAMP, steroid hormones and hyaluronic acid
* Which one is true of rate of aqueous production?
* It has a turnover of 1% per minute
* It increases during sleep
* It increases with age
* It increases during ocular inflammation
* What are the 2 major outflow pathways?
* Pressure-dependent/ TM/ conventional
* Pressure-indepdt/ Uveoscleral/ nonconventio
* What is true about facility of outflow?
* Increases with age
* Not affected by surgery, trauma, medications
* Varies widely in normal eyes, mean 0.22 to 0.28 microL/min/mmHg
* What are the 3 parts of the TM?
* Uveal, corneoscleral and juxtacanalicular
* What is false of the TM anatomy?
* TM is composed of many layers of colagenous tissue covered with endothelium
* TM has pressure-dependent flow
* TM functions as a one way valve
* The aqueous leaves the eye by bulk flow
* Laser trabeculoplasty causes apoptosis of Trabecular cells
* What is false about Schlemm’s canal?
* It is lined by endothelium and transversed by tubules
* It is a multiple channel
* Its average diameter is 370 microns
* The inner wall contains giant vacuoles that have direct communication with the intertrabecular spaces
* When IOP is low, blood may reflux into the canal
* The venous path of aqueous includes episcleral veins, anterior cyliary and superior ophthalmic veins and the cavernous sinus
* What is false about the uveoscleral outflow?
* It refers to any nontrabecular outflow
* Aqueous passes from the AC into the CBM and then into the supraciliary and suprachoroidal spaces
* It is decreased by cycloplegia, adrenergic agents, PG analogs and cyclodyalisis surgery
* It is also decreased by miotics
* It accounts for at least 5-15% of outflow

IOP and Aqueous
* What effect the following medications have on uveoscleral outflow?
* Cycloplegics
* Miotics
* Epinephrine
* Xalatan
* Brimonidine
* Timoptic ....

Clinical Evaluation
* Associate the external adnexae finding with a diagnosis associated with glaucoma
* “ash-leaf” sign
* Plexiform neuroma of upper lid
* Nevus of Ota
* Microdontia or hypodontia
* Port-wine stain (facial cutaneous angioma)
* Yellow or orange papules of skin of head/neck
* EOM restriction, proptosis, pulsating exophthalmos
* What is the type of glaucoma associated with the following corneal signs?
* Krukenberg spindle
* Exfoliating material on anterior chamber
* Keratic precipitates
* “stelate” KP’s
* “beaten bronze” appearance or corneal edema with iris changes
* What is not a possible cause of blood in Schlemm’s canal on gonioscopy
* Compression of episcleral veins with lip of goniolens
* Hypotony
* CC fistula
* Sturge-Weber syndrome
* Dilation of the pupil
* What is false about gonioscopy:
* Normal angle vessels are usually radial along the iris or circumferential on the CB
* Abnormal vessels cross the SS to reach the TM
* The nonpigmented TM is posterior to the pigmented TM
* A C angle indicates that SS is visible
* List potential causes for increased TM pigmentation:
* PDS
* PXF
* Malignant melanoma
* Trauma
* Surgery
* Hyphema

Gonioscopy
* It is false about gonioscopy
* It is necessary to see the angle due to total internal reflection at the tear-air interface (critical angle approximat 46’)
* Koeppe and Barkan lenses are examples of direct gonioscopy, most used in the OR
* Goldmann, Zeiss and Sussman lenses are used for indirect gonioscopy in the office
* Dynamic gonioscopy is done by asking the patient to move his eye
* It is false about gonioscopy
* The order of structures from anterior to posterior is: Schwalbe’s line-nonpigm TM-pigment TM-scleral spur-ciliary body-iris root
* Multiple methods of classification exist
* A Shaffer grade 4 angle is the narrowest
* Spaeth’s classification C40R indicates a normal and open angle

Clinical Evaluation
* What is angle recession and how is it different from cyclodyalisis on gonio:
* Angle recession is a tear between the longitudinal and circular muscles of CB= widened CB band
* Cyclodyalisis is a separation of the CB from the SS= gap between CB and sclera
* What is false of the Optic Nerve:
* Consists of about 1.2-1.5 million RGS axons
* The RGC cell body is in the ganglion cell layer of the retina
* The diameter of the intraocular ONH is about 2.5 mm
* There are 2 types of RGC’s: Magnocellular and Parvocellular...
OAG
* What percentage of patients with OAG have a screening IOP below 22?
* 30-50%
* What is the average corneal thickness by optical and ultrasound measurements?
* 534 optical and 544 ultrasound
* In the AGIS, patients had significantly better outcomes if their IOP was:
* Below 18 at all visits
* Below 18 50% of visits
* Below 14 at all visits
* Below 14 50% of visits....
Secondary OAG
* What is false about PXF?
* The odds of exfoliation glaucoma are 40% in 10 years
* The angle is often narrow with a +4 pigmented TM and a Sampaolesi line inferior
* The hyaline material is found on the lens, pupil margin, CB epithelium, iris PE, iris stroma, iris blood vessels and subconjunctiva
* SLE features include iris TI at pupil margin and iridodonesis or phacodonesis....
Traumatic OAG
* About hyphemas, is false:
* Sicke cell patients may have severe glaucoma from small hyphemas
* Acute IOP elevations may lead to AION or CRAO in patients with sickle cell
* Treatment consists of corticosteroids, cycloplegics, eye shield, limited activity, head elevation
* Aminocaproic acid is advocated since it has very few side effects and decreases rebleed
* Which drug is the best choice to control IOP in a hyphema patient with sickle cell diz?
* IV Diamox
* Iopidine
* Manitol
* Pilocarpine
* Timolol
OAG
* Regarding secondary glaucomas, which one is false?
* UGH syndrome can happen secondary to erosion of IOL haptic into the iris or CB
* UGH presents with chronic inflammation, iris NV, recurrent hyphemas
* Schwartz-Matsuo syndrome refers to elevated IOP after RD due to photoreceptor outer segmemts blocking of TM
* PKP related glaucomas can be of open or closed angle mechanism
* About 50% of people will have some elevation of IOP with the use of topical steroids
ACG
* About ACG epidemiology, what is incorrect?
* 10% of the 67 million cases of glaucoma worldwide are of ACG
* Predominant form of glaucoma in East Asia
* PACG is responsible for 91% of the bilateral blindness in China
* About the mechanisms of ACG, the incorrect is:
* Pupillary block is the most common cause of angle closure, including primary AC
* NVG is a type of ACG without pupillary block
* Marfan’s synd and Homocystinuria can lead to pupillary block by lens dislocation
* In Aniridia, Glaucoma is of open type since pupillary block can’t happen due to lack of iris
Primary ACG
* About the risk factors for ACG, the wrong one is:
* The prevalence varies with race: AA whites Japanese East asians Inuits
* White pts tend to have acute AC, while AA and asians have chronic asymptomatic diz
* Shallow AC, thick lens, short AL, small corneal diameter and radius increase risk
* Ages 30-50 are the highest risk
* More common in women and hyperopes

Acute PACG
* What is false about Acute PACG?
* Mild attacks can be broken with Pilocarpine 1-2%, but should avoid stronger miotics
* At IOP 40-50 the pupillary sphincter is too ischemic to respond to miotics and should use aqueous supressants, diamox or manitol
* The chance of acute attack in the fellow eye is 40-80% in 1 year
* High IOP during an acute attack may lead to ischemic nerve damage or retinal vascular occlusion

ACG
* What is false of iris and ACG?
* Both mydriasis and miosis can cause acute angle closure in a predisposed eye with shallow AC
* Miotics relax the lens zonules allowing it to sit forward in the AC, decreasing irido-lenticular touch
* Systemic medications including allergy, cold medicines, antidepressants, anticholinergics and topamax carry warning against glaucoma
* Peripheral iridectomy is indicated in patients with critically narrow angles, PAS, h/o previous attack, AC depth 2.0mm, +family history, +provocative test...
Childhood Glaucoma
* Mark the false :
* Primary congenital is the same as infantile glaucoma
* Glaucoma recognized after age 3 is termed Juvenile Glaucoma
* Glaucoma may present with buphthalmos if the IOP elevation starts after age 3
* Developmental glaucoma refers also to secondary glaucomas associated with inflammatory, neoplastic, hamartomatous, metabolic or congenital ocular or systemic anomalies
* Mark the false
* Congenital glaucoma is usually primary(50-70%)
* It is a rare disease (1:10,000 births)
* 60% are diagnosed in the first 6 months, 80% by one year
* 65% are female and bilateral in 70%
* Axenfeld-Rieger syndrome and aniridia are conditions associated with infantile glaucoma...
Medical therapy of glaucoma
* About beta blockers, which one is false:
* Betaxolol is selective for Beta 1 receptors
* They reduce aqueous production by 20-50%
* Are very additive to epinephrine
* Metipranolol was reported to cause iritis
* Effect starts within one hour and may last 4 weeks
* Tachyphylaxis and short term scape are seen
* Are contra-indicated in myasthenia gravis
Surgical Therapy
* What is false about surgical therapy of OAG?
* Usually undertaken when medical therapy fails
* LTP may be considered as a initial step in therapy since it decreases IOP by 20-25%
* The GLT showed that LTP patients did better than medication patients
* LTP is particularly effective in XFG and PG....

OKAP Glaucoma Review.ppt

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Ophthalmology Grand Rounds

A spectral-domain OCT study of formerly premature children
Presentation by: Prat Itharat MD

Grand Rounds 2007 [2] [3]
Presentation by:Marc Moore, MD

Grand Rounds 2006
Presentation by:Denise A. John

Grand Rounds 2007
Presentation by:Jeffrey D. Colburn, M.D

Surgical Outcomes in Intermittent Exotropia
Presentation by:Jeffrey D. Colburn, M.D.,

Medical Documentation
Preceptor: Louise A. Mawn, M.D
http://www.mc.vanderbilt.edu/root/ppt/eyeinstitute/Keshini%27s_ppt_5_30_08.ppt

Grand Rounds
Presentation by:Prat Itharat MD

Do You See What I See?
Presentation by:Denise A. John

Source:Vanderbilt Eye Institute

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